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1.
Environ Sci Pollut Res Int ; 30(47): 103652-103673, 2023 Oct.
Artículo en Inglés | MEDLINE | ID: mdl-37688697

RESUMEN

Particulate matter (PM) is a major primary environmental air pollutant and poses a threat to human health. Differences in the environment and leaf microstructures of plants will result in varying abilities to retain PM, but the effects of changes in these factors on PM retention are not yet well understood. This study selected 10 plant species in four urban areas (sports field, park, residential green space, and greenway) as the study objects. The amount of retained PM by the different species was measured, and the leaf microstructures were observed. It was found that the environment significantly affected both PM retention and leaf microstructure. The ranking of PM retention in the 10 species in four areas was greenway > residential green space > park > sports field. The ranking of average stomatal width and length was park > sports field > residential green space > greenway, while that of average stomatal density was greenway > residential green space > park > sports field. Different environments affected the length and density of trichomes in the leaves. These changes represented the adaptation of plant species to the growth environment. The stomata and grooves of the leaf surface significantly affected the ability of plants to retain PM. The amount of PM retained by different species varied. In all four urban areas, Prunus × cistena N. E. Hansen ex Koehne (purple leaf sand cherry), Prunus cerasifera Ehrhart f. atropurpurea (Jacq.) Rehd. (cherry plum), Buxus sinica var. parvifolia M. Cheng (common boxwood), and Ligustrum × vicaryi Rehder (golden privet) showed strong PM retention. The results of this study will provide information for planners and urban managers for the selection of plant species.


Asunto(s)
Contaminantes Atmosféricos , Material Particulado , Humanos , Material Particulado/análisis , Monitoreo del Ambiente/métodos , Contaminantes Atmosféricos/análisis , Plantas , Hojas de la Planta/química , Árboles/química
2.
Environ Pollut ; 293: 118560, 2022 Jan 15.
Artículo en Inglés | MEDLINE | ID: mdl-34808309

RESUMEN

The effects of air pollution on adolescents need further consideration. Although there is evidence that maternal exposure to air pollution may affect the cognitive function of offspring, relevant studies remain limited and inconsistent, with a lack of studies assessing the causal effects and evidence from developing countries. Using data from Chinese Family Panel Studies, a representative Chinese nationwide cohort study, OLS combined with instrumental variable + two-stage least square (IV+2SLS) was used to explore the causal effects of exposure to PM2.5 concentrations during pregnancy on the cognitive function of offspring when they become adolescents. After detailed argumentation and multiple testing, Planetary Boundary Layer Height (PBLH) and Surface Pressure (SP) were selected as the instrumental variables for this study. One thousand five hundred fifty-five adolescents participated in this study, with a mean age of 13.3 years (sd = 2.3). There were 706 females (45.4%), the mean maternal PM2.5 exposure concentration was 64.9 µg/m3, and recorded a mean cognitive function score of 38.1 (sd = 9.4). The OLS results found that maternal exposure to air pollution increased cognitive function in offspring adolescents, corroborating the presence of endogeneity. Multi-domain knowledge, the results of the weak instrumental variable assessments of F-tests (F = 237 > 10) and Stock-yogo tests (minimum eigenvalue statistic = 153.16 > 16.38), and the results of the Hansen J overidentification test (p > 0.05) verified the plausibility and validity of the instrumental variables. The IV+2SLS results, following causal modeling, showed that PM2.5 exposure during pregnancy impairs the cognitive ability of offspring adolescents (ß = -0.040, p < 0.05). Robustness tests also validated the results. This study provides important policy implications for developing countries on protecting their adolescents and reminds parents that the protection of adolescents from air pollution should begin from conception.


Asunto(s)
Contaminantes Atmosféricos , Material Particulado , Adolescente , Contaminantes Atmosféricos/análisis , China , Cognición , Estudios de Cohortes , Femenino , Humanos , Material Particulado/análisis , Embarazo
3.
Indian J Dermatol Venereol Leprol ; 83(4): 415-423, 2017.
Artículo en Inglés | MEDLINE | ID: mdl-28195077

RESUMEN

The increase in air pollution over the years has had major effects on the human skin. Various air pollutants such as ultraviolet radiation, polycyclic aromatic hydrocarbons, volatile organic compounds, oxides, particulate matter, ozone and cigarette smoke affect the skin as it is the outermost barrier. Air pollutants damage the skin by inducing oxidative stress. Although human skin acts as a biological shield against pro-oxidative chemicals and physical air pollutants, prolonged or repetitive exposure to high levels of these pollutants may have profound negative effects on the skin. Exposure to ultraviolet radiation has been associated with extrinsic skin aging and skin cancers. Cigarette smoke contributes to premature aging and an increase in the incidence of psoriasis, acne and skin cancers. It is also implicated in allergic skin conditions such as atopic dermatitis and eczema. Polyaromatic hydrocarbons are associated with extrinsic skin aging, pigmentation, cancers and acneiform eruptions. Volatile organic compounds have been associated with atopic dermatitis. Given the increasing levels of air pollution and its detrimental effects on the skin, it is advisable to use strategies to decrease air pollution.


Asunto(s)
Contaminantes Atmosféricos/efectos adversos , Contaminación del Aire/efectos adversos , Envejecimiento de la Piel/patología , Piel/patología , Rayos Ultravioleta/efectos adversos , Humanos , Material Particulado/efectos adversos , Hidrocarburos Policíclicos Aromáticos/efectos adversos , Neoplasias Cutáneas/inducido químicamente , Neoplasias Cutáneas/etiología , Neoplasias Cutáneas/patología
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